• 021-34922321-2 || admissions@sohailuniversity.edu.pk
  • gulnaz@sohailuniversity.edu.pk
  • Department ofBiomedical & Biological Sciences
  • 0300-00000
  • Ext:000

Gulnaz Khan

Lecturer

Education
PhD (Continue), Genetics, University of Karachi
MPhil (2018), Molecular Medicine, Dr. Panjwani Center for Molecular Medicine & Drug Research, International Center for Chemical & Biological Sciences, University of Karachi
MSc (2014), Biotechnology, University of Karachi

Experience
Lecturer (2019 to present), Department of Biomedical & Biological Sciences, Sohail University
Manager (2018 to present), Biomedical Research Center, Sohail University
PhD Scholar (2018 to present), Genetics, University of Karachi
Research Scholar (2015-2018), Dr. Panjwani Center for Molecular Medicine & Drug Research, International Center for Chemical & Biological Sciences, University of Karachi

Research interest
My research is focused on mechanistic studies involving the development of non-communicable diseases particularly metabolic disorders and cancer. Previously, I explored the mechanism of natural/synthetic compounds in the treatment of metabolic disorders on cellular and molecular level. I am also directing my motives to perceive genetic mechanism of these pathologies in relation to advance glycation end products.

Keywords
Advanced glycation end product, inflammation, metabolic disorders, reactive oxygen species, signal transduction.

Publications
Khan, Gulnaz, Meha Fatima Aftab, Bilquees Bano, Khalid Mohammed Khan, Munazza Murtaza, Sonia Siddiqui, M. Hafizur Rehman, and Rizwana Sanaullah Waraich. “A new indanedione derivative alleviates symptoms of diabetes by modulating RAGE-NF-kappaB pathway in db/db mice.” Biochemical and biophysical research communications 501, no. 4 (2018): 863-870. (ISI, IF: 2.559)
Murtaza, Munazza, Gulnaz Khan, Meha Fatima Aftab, Shabbir Khan Afridi, Safina Ghaffar, Ayaz Ahmed, Rahman M. Hafizur, and Rizwana Sanaullah Waraich. “Cucurbitacin E reduces obesity and related metabolic dysfunction in mice by targeting JAK-STAT5 signaling pathway.” PloS one 12, no. 6 (2017): e0178910. (ISI, IF: 2.766)